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BUT JTR . . . (Hair Multiplication & Research)

posted by John The Revelator, 16.09.2007, 19:56

» BTW wouldn't there be more to balding than testosterone levels?
Of course, but testosterone levels determine how much DHT can be in circulation, because testosterone is converted into DHT by the reductase enzyme. So if you have genetic receptor sensitivity to DHT, and high testosterone levels, you'll express MPB. But if you have genetic receptor sensitivity plus very low testosterone levels (as in females or most males of advanced age), even with the genetics in place, you'll probably express MPB very weakly.

» Sme men don't go bald until they are middle aged--a time when they have
» less testosterone than when they were younger (one possible reason: it
» would be a combo of how much over how long that causes hair to fall out,
» and that could vary from person to person).
Yes, it's a combination of a lot of things:

1) The testosterone levels in your body, which determine DHT levels.

2) Your genes -- genetics determine the sensitivity of your cell receptors to DHT. So, lots of men who have huge testosterone levels, and thus huge DHT levels, still don't go bald because they have great genetics.

3) Your genetics also determines the AGE at which your genetically-determined receptor sensitivity to DHT kicks in, if at all. So, in some men it's 18, in others it's 25, in others it may be 35 or 50.

In general, a general rule is that, apart from senescent baldness, the later in life the receptor sensitivity genes kick in, the LOWER the man's ultimate Norwood level will be when it stabilizes.

So, if a man's receptor sensitivity MPB genes kick in at 15, there's a much higher chance he'll stabilize at a NW 7. But if his receptor sensitivity genes kick in much later, say at 40, then there's a much higher chance that he'll stabilize at, say, NW 2 or 3.

This is largely (but not only) because, as he gets older, his testosterone levels are declining, so less DHT is being produced.


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