Hair Loss Forum - Answer this Poll - requested by Boyinhtown

March 2017 - Trending Topics in our new forum

 Stem cell hair restoration results - Dr. Cole vs Histogen vs RepliCel.
 Dr. Paul Kemp, CEO of HairClone answers questions about follicle banking.
 RepliCel year 2017 forecast for RCH-01 cell based hair regeneration.
 Dr. Cole to start stem cell hair restoration trial in the US.
 Dr. Umar's 6500 grafts life saving repair procedure for transplant victim.
 FUE donor harvesting, what you must know when choosing a clinic.
 Dr. Koray Erdogan raises the standard in FUE artistry skills.
 Dr. Arvind on how to formulate your own toxic free shampoo.
 Conference & FREE hair restoration consultations; cities worldwide.

This is a READ ONLY forum.
Access our brand new platform at HairSite New Forum to continue the discussion on these topics that you are interested in. All contents and discussions have been transferred to our new forum at
  HairSite New Forum

Log in | User | Register

Thread view  Order  «  
 

gutted

20.05.2012, 18:49
(edited by gutted, 20.05.2012, 19:05)

@ KO

Answer this Poll - requested by Boyinhtown

» » this can be explained by the distribution of type 2, it can be assumed
» » type2 is more prominent in the body and leads to a more systemic
» » introduction of dht into the blood stream hence inhibiting type 2 leads
» to
» » a more pronounced effect on the dht levels in the blood stream, which
» means
» » the sebaceous gland isnt hyperactive as much, THUS allows the
» body
» » to disinfect the infected cells.
»
»
» » Inhibiting type 1 probably doesnt result in a major decerease in serum
» dht
» » levels which is why it never helped in aiding the body in getting rid of
» » the infection in tha mk386 study. Also bear in mind IGF1 is also a
» » stimulator of the sebgland.
» »
» NOT TRUE. Serum DHT levels are reduced by MK386.
»
» http://jcem.endojournals.org/content/82/5/1373.short

Ok, but not to a level to help the body wipe the infection. - You also need to take into to account igf1/testosterone levels as these all stimulate the sebgland

According to my theory Hair miniturisation does not occur through androgens binding to DP AR.
It occurs because there is a chronic micro infection of the follcile/counterparts which the body is battling and which appears to fight off this infection with the help of fin, but

1) either somehwhere leaves behind damaged cells

OR

2)either these cells, during the regeneration stage in the hair cycle, the microinfection abruptly returns, preventing the regeneration of a thick hair.
(people who reportedley experience, mintuarised hair turning thicker are those people that have successfully wiped the chronic microinfection)

OR

3) the infection is not fully controlled, its still there in some cells, and is preventing full regeneration.

So basically what im trying to say is baldness is a chronic microinfection of the follcile/counterparts.

So if with fin/mk386, homestasis occurs in an indivdual, the body's androgen levels return to high levels, this in turn causes infection and means people experience the so called "mpb advancing" with the anagen hair cycle shortneing and subsequent mintuarisation. This is NOT fin losing effectiveness.

Similarly if homestasis is not achieved and dht levels stay controlled - fin wil work to grow hair because it lowers dht in the body alowing the body to fight the infection off.

Terminal to Vellus hairs are the result of those hair/counterpart cells shutting down production - similar to what happens in the body when cancer is detected.




gutted is located in [NA] and he is available to meet: NO


Post reply
gutted

20.05.2012, 21:11

@ KO

Answer this Poll - requested by Boyinhtown

» » this can be explained by the distribution of type 2, it can be assumed
» » type2 is more prominent in the body and leads to a more systemic
» » introduction of dht into the blood stream hence inhibiting type 2 leads
» to
» » a more pronounced effect on the dht levels in the blood stream, which
» means
» » the sebaceous gland isnt hyperactive as much, THUS allows the
» body
» » to disinfect the infected cells.
»
»
» » Inhibiting type 1 probably doesnt result in a major decerease in serum
» dht
» » levels which is why it never helped in aiding the body in getting rid of
» » the infection in tha mk386 study. Also bear in mind IGF1 is also a
» » stimulator of the sebgland.
» »
» NOT TRUE. Serum DHT levels are reduced by MK386.
»
» http://jcem.endojournals.org/content/82/5/1373.short


this is taken from - regrowth com/hairloss-remedy/mk_386/mk_386.cfm

"MK-386 is a relatively new antiandrogen that has been shown to inhibit ONLY type 1 DHT. It inhibits circulating DHT by 20-30%. In combination with Proscar, it reduced DHT by an average of 89%, compared with 68% by Proscar alone!"

If that is true mk386 doesnt even touch circulating androgens!

This is taken from the study you linked to -

"Treatments were given once daily for 14 days, except in 1 panel in which MK-386 was administered 10 mg twice daily for comparison to 20 mg daily. Serum, sebum, and semen DHT concentrations and serum and sebum T concentrations were measured before and after treatment. "

14 days is hardly a good time frame to judge efficacy on hair growth!!




gutted is located in [NA] and he is available to meet: NO


Post reply
Ahab

21.05.2012, 00:36

@ Bryan

Your Answer

» I'll ask you the same question I asked somebody else in this thread: how
» do you explain the beneficial effect on hair growth from finasteride and
» dutasteride? :-)

Those drugs reduce the effect of DHT.

Perhaps certain nutrients also reduce the effect of DHT.




Ahab is located in [NA] and he is available to meet: NO


Post reply
Bryan

21.05.2012, 05:52

@ Bryan

Answer this Poll - requested by Boyinhtown

I can't help but notice, "gutted", that you haven't made any reply to me since early yesterday. I can imagine that you may have been a little confused by what I said about differences in the effects of MK386 and finasteride, so you tried to blame that on supposed effects on sebum production. If that's the case, then let's avoid altogether the complicated issue of 5a-reductase inhibition, and stick with just the direct effect of androgens on hair follicles! Androgens suppress the growth of scalp hair follicles, and they do it both in vivo and in vitro; since that's the OPPOSITE of what happens with most body hair, that proves that there's a difference in the design of scalp hair follicles (because of genetics, evidently), compared to most body hair (like hair follicles in the mustache). I had to laugh when you said in an ealier post that "DHT and androgens are required for the growth of hair", because that's the LEAST scientific thing I can imagine anybody ever saying about hair loss! It's fine to speculate along the lines that sebum might have SOMETHING to do with balding (by way of the immune system), but it becomes simply laughable to say that DHT is required for scalp hair to grow! :-)




Bryan is located in [NA] and he is available to meet: NO


Post reply
gutted

21.05.2012, 13:37

@ Bryan

Answer this Poll - requested by Boyinhtown

» I can't help but notice, "gutted", that you haven't made any reply to me
» since early yesterday. I can imagine that you may have been a little
» confused by what I said about differences in the effects of MK386 and
» finasteride, so you tried to blame that on supposed effects on sebum
» production.

which post was that?

I explained with mk386 - it doesnt matter which enzyme is inhibited, type 1 or type 2. Type 2 is more prominent and feeds the body with probably the most the dht (systemic and tissue alike).
Type one probably feeds the body with a 1/3 - inhibiting a 1/3 (with mk386) of the dht most likley never helped the body (immune system) in getting rid of this microinfection that appears - you need to think of it like this - if the body is trying to get rid of an infection but dht/igf1/testosterone, fluctuate to high levels, pumping out whatever it is feeding the bacteria causing overgrowth at the same time, its being prevented from totally removing that infection....we need a balance of the 2 scales, this is what fin does in some lucky individuals.

The study was 14 days long? how does anyone expect any benneficial effects/or lack of on hair in this time frame?
According to my thoery - testosterone/igf1 ALSO needs to be taken into account.

If that's the case, then let's avoid altogether the
» complicated issue of 5a-reductase inhibition, and stick with just the
» direct effect of androgens on hair follicles! Androgens suppress
» the growth of scalp hair follicles, and they do it both in vivo and
» in vitro;
since that's the OPPOSITE of what happens with most body
» hair, that proves that there's a difference in the design of scalp
» hair follicles (because of genetics, evidently), compared to most body hair
» (like hair follicles in the mustache).

I already explained the study MAY have been flawed with reasons outlined in my ealrier post.
The sebgland may have already been infected OR contain those bacteria and immune cells may have also been present surpressing the growth upon administration of andorgens.

Androgens do not surpress growth according to my thoery - this, hopefully in sometime in th future will be proven.
They may indirectly surpress growth via an increase in bacteria but accoring to my theory they do not surpress growth directly via DP AR.

»I had to laugh when you said in an
» ealier post that "DHT and androgens are required for the growth of hair",
» because that's the LEAST scientific thing I can imagine anybody ever saying
» about hair loss! It's fine to speculate along the lines that sebum might
» have SOMETHING to do with balding (by way of the immune system), but it
» becomes simply laughable to say that DHT is required for scalp hair
» to grow! :-)

whether they are required or not required for growth, doesnt matter - they may be needed to maintain the hair cycle or may not be (im not aware of the facts) - but what i do say is androgens have NO DIRECT, NEGATIVE effects on hair, according to my theory of course.




gutted is located in [NA] and he is available to meet: NO


Post reply
gutted

21.05.2012, 17:20

@ Bryan

Answer this Poll - requested by Boyinhtown

» Androgens suppress
» the growth of scalp hair follicles, and they do it both in vivo and
» in vitro; since that's the OPPOSITE of what happens with most body
» hair, that proves that there's a difference in the design of scalp
» hair follicles (because of genetics, evidently), compared to most body hair
» (like hair follicles in the mustache).

i explained in an earlier post, genetics is probably also a component to why some people go bald and others dont, but certainly not the genetics of the AR gene or individual hair follciles themselves which current science states.

The genetics part can be explained by various ways.

The distribution/concentration of the the sebglands/sweat glands between the donor site and the balding site, with a higher concentration/distribution in the balding site.

Sebgland/sweat gland distribution/concentration is genetically determined when you are born. People who dont go bald are those that are lucky to have a low distribution/concentration of those glands in the scalp.




gutted is located in [NA] and he is available to meet: NO


Post reply
Iron_Man 

Losers Nightmares,
21.05.2012, 20:45

@ gutted

Answer this Poll - requested by Boyinhtown

Follicles need DHT to grow!! Stop this nonsense that DHT causes hair loss




Iron_Man  is located in LOSERS NIGHTMARES and he is available to meet: NO

---
HAIRSITE READERS BEWARE!
HairSite and this forum software allows the registration of identical usernames!
Read more: Iron_Man vs. Iron_Man

BEWARE HairSite readers! Don’t feed the TROLLS, SHILLS & SOCK PUPPETS and PSYCHO’s - because this is my job!


Post reply
Bryan

21.05.2012, 21:07

@ gutted

Answer this Poll - requested by Boyinhtown

» I explained with mk386 - it doesnt matter which enzyme is inhibited, type 1
» or type 2. Type 2 is more prominent and feeds the body with probably the
» most the dht (systemic and tissue alike).
» Type one probably feeds the body with a 1/3 - inhibiting a 1/3 (with mk386)
» of the dht most likley never helped the body (immune system) in getting rid
» of this microinfection that appears - you need to think of it like this -
» if the body is trying to get rid of an infection but dht/igf1/testosterone,
» fluctuate to high levels, pumping out whatever it is feeding the bacteria
» causing overgrowth at the same time, its being prevented from totally
» removing that infection....we need a balance of the 2 scales, this is what
» fin does in some lucky individuals.

Systemic levels of DHT (DHT in the bloodstream, in other words) have relatively little effect on hair follicles or sebaceous glands. The type 2 and type 1 forms of 5a-reductase affect those structures when they actually contain those forms of 5a-reductase. Your explanation that MK386 has no effect on hair because of only a relatively minor effect on DHT levels in the blood is just an excuse. It's dead WRONG.

» Androgens do not surpress growth according to my thoery - this, hopefully
» in sometime in th future will be proven.
» They may indirectly surpress growth via an increase in bacteria but
» accoring to my theory they do not surpress growth directly via DP AR.
»
» » I had to laugh when you said in an earlier post that
» » "DHT and androgens are required for the growth of hair",
» » because that's the LEAST scientific thing I can imagine anybody
» » ever saying about hair loss! It's fine to speculate along the
» » lines that sebum might have SOMETHING to do with balding (by way
» » of the immune system), but it becomes simply laughable to say
» » that DHT is required for scalp hair to grow! :-)
»
» whether they are required or not required for growth, doesnt matter - they
» may be needed to maintain the hair cycle or may not be (im not aware of the
» facts) - but what i do say is androgens have NO DIRECT,
» NEGATIVE effects on hair, according to my theory of course.

You need to stop paying attention to your THEORY, and start going by the FACTS of the matter. Doctors and scientists have soundly PROVEN that androgens have a direct negative effect on scalp hair follicles. This isn't something controversial, it's common knowledge! It's time for you to become familiar with the science of this matter, and not simply go around repeating things that have to do with your silly theory.




Bryan is located in [NA] and he is available to meet: NO


Post reply
Bryan

21.05.2012, 21:26

@ Iron_Man 

Answer this Poll - requested by Boyinhtown

» Follicles need DHT to grow!! Stop this nonsense that DHT causes hair loss

You're completely, totally cuckoo.




Bryan is located in [NA] and he is available to meet: NO


Post reply
gutted

21.05.2012, 21:49

@ Bryan

Answer this Poll - requested by Boyinhtown

» » I explained with mk386 - it doesnt matter which enzyme is inhibited, type
» 1
» » or type 2. Type 2 is more prominent and feeds the body with probably the
» » most the dht (systemic and tissue alike).
» » Type one probably feeds the body with a 1/3 - inhibiting a 1/3 (with
» mk386)
» » of the dht most likley never helped the body (immune system) in getting
» rid
» » of this microinfection that appears - you need to think of it like this
» -
» » if the body is trying to get rid of an infection but
» dht/igf1/testosterone,
» » fluctuate to high levels, pumping out whatever it is feeding the
» bacteria
» » causing overgrowth at the same time, its being prevented from totally
» » removing that infection....we need a balance of the 2 scales, this is
» what
» » fin does in some lucky individuals.
»
» Systemic levels of DHT (DHT in the bloodstream, in other words) have
» relatively little effect on hair follicles or sebaceous glands. The type 2
» and type 1 forms of 5a-reductase affect those structures when they actually
» contain those forms of 5a-reductase. Your explanation that MK386
» has no effect on hair because of only a relatively minor effect on DHT
» levels in the blood is just an excuse. It's dead WRONG.
»


I said systemic AS WELL AS tissue levels of dht.
Im not denying DHT is a factor in hairloss - BUT an indirect factor.


» » Androgens do not surpress growth according to my thoery - this,
» hopefully
» » in sometime in th future will be proven.
» » They may indirectly surpress growth via an increase in bacteria but
» » accoring to my theory they do not surpress growth directly via DP AR.
» »
» » » I had to laugh when you said in an earlier post that
» » » "DHT and androgens are required for the growth of hair",
» » » because that's the LEAST scientific thing I can imagine anybody
» » » ever saying about hair loss! It's fine to speculate along the
» » » lines that sebum might have SOMETHING to do with balding (by way
» » » of the immune system), but it becomes simply laughable to say
» » » that DHT is required for scalp hair to grow! :-)
» »
» » whether they are required or not required for growth, doesnt matter -
» they
» » may be needed to maintain the hair cycle or may not be (im not aware of
» the
» » facts) - but what i do say is androgens have NO DIRECT,
» » NEGATIVE effects on hair, according to my theory of course.
»
» You need to stop paying attention to your THEORY, and start going by the
» FACTS of the matter. Doctors and scientists have soundly PROVEN that
» androgens have a direct negative effect on scalp hair follicles. This
» isn't something controversial, it's common knowledge! It's time for you to
» become familiar with the science of this matter, and not simply go
» around repeating things that have to do with your silly theory.

And you need to pay attention to the possibility that some of those facts may be flawed!

Pre programmed minituarisation is simply a myth based on potentially flawed science, that really does need to be looked at again.




gutted is located in [NA] and he is available to meet: NO


Post reply
Bryan

21.05.2012, 22:01

@ gutted

Answer this Poll - requested by Boyinhtown

» And you need to pay attention to the possibility that some of those
» facts may be flawed!

The fact that androgens DIRECTLY affect scalp hair follicles isn't "flawed". It's common knowledge. You need to start reading what doctors and scientists say, and stop paying attention to your "theory".




Bryan is located in [NA] and he is available to meet: NO


Post reply
gutted

21.05.2012, 22:14

@ Bryan

Answer this Poll - requested by Boyinhtown

» » And you need to pay attention to the possibility that some of those
» » facts may be flawed!
»
» The fact that androgens DIRECTLY affect scalp hair follicles isn't
» "flawed". It's common knowledge. You need to start reading what doctors
» and scientists say, and stop paying attention to your "theory".

yes...possibly flawed, "common" knowlege.




gutted is located in [NA] and he is available to meet: NO


Post reply
Iron_Man 

Losers Nightmares,
21.05.2012, 22:24

@ Bryan

Answer this Poll - requested by Boyinhtown

» » Follicles need DHT to grow!! Stop this nonsense that DHT causes hair
» loss
»
» You're completely, totally cuckoo.

then how do you explain the fact that Gho's procedure cures hair loss permanently 100%




Iron_Man  is located in LOSERS NIGHTMARES and he is available to meet: NO

---
HAIRSITE READERS BEWARE!
HairSite and this forum software allows the registration of identical usernames!
Read more: Iron_Man vs. Iron_Man

BEWARE HairSite readers! Don’t feed the TROLLS, SHILLS & SOCK PUPPETS and PSYCHO’s - because this is my job!


Post reply
gutted

21.05.2012, 22:28

@ Iron_Man 

Answer this Poll - requested by Boyinhtown

» » » Follicles need DHT to grow!! Stop this nonsense that DHT causes hair
» » loss
» »
» » You're completely, totally cuckoo.
»
» then how do you explain the fact that Gho's procedure cures hair loss
» permanently 100%

2020 stop trolling as iron man!




gutted is located in [NA] and he is available to meet: NO


Post reply
KO

21.05.2012, 23:41

@ Bryan

Answer this Poll - requested by Boyinhtown

» » Follicles need DHT to grow!! Stop this nonsense that DHT causes hair
» loss
»
» You're completely, totally cuckoo.

Bryan, just give up. For some reason he is obsessed with his theory, and I can't spot why.




KO is located in [NA] and he is available to meet: NO


Post reply
Bryan

22.05.2012, 03:47

@ KO

Answer this Poll - requested by Boyinhtown

» » » Follicles need DHT to grow!! Stop this nonsense that DHT causes
» » » hair loss
» »
» » You're completely, totally cuckoo.
»
» Bryan, just give up. For some reason he is obsessed with his theory, and I
» can't spot why.

I think you're right, KO! After you explain repeatedly what doctors and scientists say on this issue and all they do is keep repeating the same tired things over and over and over, it's time just to give it up! :-)




Bryan is located in [NA] and he is available to meet: NO


Post reply
gutted

22.05.2012, 13:23
(edited by gutted, 22.05.2012, 13:44)

@ Bryan

Answer this Poll - requested by Boyinhtown

» » » » Follicles need DHT to grow!! Stop this nonsense that DHT causes
» » » » hair loss
» » »
» » » You're completely, totally cuckoo.
» »
» » Bryan, just give up. For some reason he is obsessed with his theory, and
» I
» » can't spot why.
»
» I think you're right, KO! After you explain repeatedly what doctors
» and scientists say on this issue and all they do is keep repeating the same
» tired things over and over and over, it's time just to give it up! :-)

I understand and fully take on board what doctors and scientists say. I never disputed this, i only asked for it to be acknowledged that this "androgens binding to the dermal papilla AR causing minuturisation/anagen cycl shortening, in Vitro" study has the potential to be flawed and needs to be looked at again - of course this is not only based on my anecdotal experience but based on certain other studies such as -

certain antibiotics causing hair re-growth - people probably assume the hair growth properties are due to a secondary unknown mechanism BUT this may not be the case and it could be the traditional infection killing properties antibiotics are known for.

Certain anti inflammotaries causing hair regrowth.

These so called "scientific facts" need to be looked at again as i 1000% believe (but obviously cannot prove) there is no such thing as hair is "destined" to go bald, it is because of damage caused by the immune system because of micro-infection that causes hair cells damage and eventually cause them to shut down production of a hair i.e terminal to vellus.

Fin helps indirectly by lowering androgens in the body, be it tissue/serum/systemic androgens, for the body to clear the infection causing thinning hair to thicken back up BUT not once hair cells have shut down production.

Heres something you may laugh at too bryan -

Fin does NOT need to be taken on a continuous basis, it only needs to be taken for the duration the infection is present to help the body wipe the infection.

But once users get off fin "Destined, Catch up baldness" (no such thing) is percieved because once a person gets off fin, over many many years - slowly but steadily, homestasis was taking its course during the users time on fin, causing a slow but steady rise in the infection spreading and clearing again.
Once a user totally gets off the drug - this causes a massive spike in tissue/systemic androgen levels causing the knock on effect on major reinfection starting and spreading and clearing again, thus further advancing baldness(hair minutrision/anagen hair cycle shortneing which eventually causes certain cells to shut down production of a hair i.e terminal to vellus) at a very fast pace - THIS is why "catch up baldness" is percieved!

Baldness can be distilled down to a micorinfection that results because of unaccepatable levels of androgens/igf1/other sebgland/sweat gland modulators fuelling this infection and reinfection.

All that is needed to treat (not to cure) a person of baldness is to periodically use an antibiotic THIS is what fin indirectly allows the body to do!

There are reports antibiotics cause hair loss in individuals - this is NOT hair loss, this is the antibiotic clearing the infection at the hair follcile cells i.e hair shedding, which also sometimes occurs with fin.

Nizoral/tgel was used in the merck study for a reason!

Of course this is not proven and is my own theory.




gutted is located in [NA] and he is available to meet: NO


Post reply
hairman2

22.05.2012, 13:45

@ gutted

Answer this Poll - requested by Boyinhtown

» These so called "scientific facts" need to be looked at again as i 1000%
» believe (but obviously cannot prove) there is no such thing as hair is
» "destined" to go bald, it is because of damage caused by the immune system
» because of micro-infection that causes hair cells damage and
» eventually cause them to shut down production of a hair i.e terminal to
» vellus.

no one cares in personal belief if it cannot be proven. You can believe in Mohammed, Jesus or the flying spaghetti Monster.. without proof it has no value




hairman2 is located in [NA] and he is available to meet: NO


Post reply
gutted

22.05.2012, 13:51

@ hairman2

Answer this Poll - requested by Boyinhtown

» » These so called "scientific facts" need to be looked at again as i
» 1000%
» » believe (but obviously cannot prove) there is no such thing as hair
» is
» » "destined" to go bald, it is because of damage caused by the immune
» system
» » because of micro-infection that causes hair cells damage and
» » eventually cause them to shut down production of a hair i.e terminal to
» » vellus.
»
» no one cares in personal belief if it cannot be proven. You can believe in
» Mohammed, Jesus or the flying spaghetti Monster.. without proof it has no
» value

i agree with you there.




gutted is located in [NA] and he is available to meet: NO


Post reply
Bryan

22.05.2012, 19:52

@ gutted

Answer this Poll - requested by Boyinhtown

» I understand and fully take on board what doctors and scientists say. I
» never disputed this, i only asked for it to be acknowledged that this
» "androgens binding to the dermal papilla AR causing minuturisation/anagen
» cycl shortening, in Vitro" study has the potential to be flawed and needs
» to be looked at again - of course this is not only based on my anecdotal
» experience but based on certain other studies such as - [snip]

Top-notch hairloss researchers have all shown the exact same thing: when androgens are given to either humans or other balding primates (like stumptailed macaques), the same thing happens: the growth of those scalp hair follicles is suppressed. The exact OPPOSITE of that happens when body hair follicles are used, instead: growth is actually STIMULATED by androgens. The same thing also happens when the hair follicles are removed from the body altogether, and put in a test tube or petri dish: scalp follicles are suppressed by androgens, while body hair follicles are stimulated by those same androgens.

You need to face the scientific facts, and stop harping on this silly idea that "infections" are what affect hair growth. Bacteria don't affect hair growth when follicles are sitting in a petri dish.




Bryan is located in [NA] and he is available to meet: NO


Post reply
Iron_Man 

Losers Nightmares,
22.05.2012, 21:10

@ Bryan

Answer this Poll - requested by Boyinhtown

» You need to face the scientific facts, and stop harping on this silly idea
» that "infections" are what affect hair growth. Bacteria don't affect hair
» growth when follicles are sitting in a petri dish.

^ this.

It's not an infection. It's not your immune system and it's not your diet. Please stop posting this nonsense.


Message me so I could send you a coupon for 10% for Dr.Gho's services.

Dr.Gho - the real cure to hair loss!




Iron_Man  is located in LOSERS NIGHTMARES and he is available to meet: NO

---
HAIRSITE READERS BEWARE!
HairSite and this forum software allows the registration of identical usernames!
Read more: Iron_Man vs. Iron_Man

BEWARE HairSite readers! Don’t feed the TROLLS, SHILLS & SOCK PUPPETS and PSYCHO’s - because this is my job!


Post reply
gutted

22.05.2012, 21:33

@ Bryan

Answer this Poll - requested by Boyinhtown

» » I understand and fully take on board what doctors and scientists say. I
» » never disputed this, i only asked for it to be acknowledged that this
» » "androgens binding to the dermal papilla AR causing
» minuturisation/anagen
» » cycl shortening, in Vitro" study has the potential to be flawed and
» needs
» » to be looked at again - of course this is not only based on my anecdotal
» » experience but based on certain other studies such as - [snip]
»
» Top-notch hairloss researchers have all shown the exact same thing: when
» androgens are given to either humans or other balding primates (like
» stumptailed macaques), the same thing happens: the growth of those scalp
» hair follicles is suppressed. The exact OPPOSITE of that happens when
» body hair follicles are used, instead: growth is actually
» STIMULATED by androgens. The same thing also happens when the hair
» follicles are removed from the body altogether, and put in a test tube or
» petri dish: scalp follicles are suppressed by androgens, while body hair
» follicles are stimulated by those same androgens.
»
» You need to face the scientific facts, and stop harping on this silly idea
» that "infections" are what affect hair growth. Bacteria don't affect hair
» growth when follicles are sitting in a petri dish.

Bacteria ARE natrually present in the hair follicle. So it can be assumed Bacteria WAS indeed present in the dissected follcile.

I think based on my observations im already convinced that bacteria are the cause of mpb, and nothing you or anyone else says will convince me that androgens cause baldness/hair minutuarisation.

I do state however, this is NOT PROVEN just as a disclaimer for the, naive, hair loss sufferers that may be reading this, which there are plenty of!

I on the other hand will be proactive rather than take in flawed science! :)




gutted is located in [NA] and he is available to meet: NO


Post reply

Thread view  Order  «  
 
120100 Postings in 12546 Threads, 6047 registered users
Hair Loss Forum | Admin contact

 
This is a READ ONLY forum.
Access our brand new platform at HairSite New Forum to continue the discussion on these topics. All contents and discussions have been transferred to
HairSite New Forum

Disclosure: This is an advertising site for our paid sponsors & advertisers. The contents, videos & photos on HairSite are provided by paid sponsors and are not endorsed by HairSite in any way. The recommendations, results, and representations made by our sponsors/advertisers do not reflect the opinions of HairSite. This site is to showcase successful hair restoration results only. It is not the mandate of this site to engage in the discussion of failed, unsuccessful procedures, lawsuits, litigations or complaint cases; comments of such nature, including external links, may be removed from the forum. Notify hairsite@aol.com any false, defamatory, misleading or inappropriate user generated contents for immediate removal from the forum. Also read Terms of Use & Privacy Statement |  HairSite advertisers: ASMED | Dr. Bhatti | Dr. Bisanga | Dr. Cole | Dr. Hakan Doganay | Dr.Epstein | Dr. Jones | Dr. Halder | Hasson & Wong | Dr. Klein | Dr. Madhu | Dr. Mwamba | Dr. Donald Ng| Dr. Poswal | Dr. Rahal | Dr. Razack | Dr. Reddy | Dr. Umar | Dr. Woods | DHI Global | HDC Clinic | |Lasercomb | Reviva Clinic | Ziering Medical|